“The irrational fear of sugar”
Glucose / C6H12O6
a monosaccharide (or simple sugar), is an important carbohydrate in mammalian biology. The living cell uses it as a source of energy and metabolic intermediate. Glucose is one of the main products of photosynthesis and starts cellular respiration in both prokaryotes and eukaryotes. The name comes from the Greek word glykys (γλυκύς), which means "sweet", plus the suffix "-ose" which denotes a carbohydrate.
Insulin
(from Latin insula, "island", as it is produced in the Islets of Langerhans in the pancreas) is an anabolic polypeptide hormone that regulates blood plasma glucose levels and the availability and entry of macro nutrients to the cellular mitochondria. Insulin is composed of 51 amino acid residues and has a molecular weight of 5808. Insulin's structure varies slightly between animal species. Apart from being the primary agent in carbohydrate homeostasis, it has effects on amino acid & fat metabolism as well as changes the liver's activity in storing or releasing glucose and in processing blood lipids. Insulin exerts a strong effect on other tissues such as fat and muscle. The amount of insulin in circulation has extremely widespread effects throughout the body.
The actions of insulin on cells include
translocation of Glut-4 transporter to the plasma membrane and influx of glucose, glycogen synthesis, glycolysis and fatty acid synthesis
Control of cellular intake of substrates, most prominently glucose in muscle and adipose tissue.
Increase of DNA replication and protein synthesis via control of amino acid uptake.
Modification of the activity of numerous enzymes (allosteric effect).
Increased glycogen synthesis – insulin facilitates the entry of glucose to the liver (and muscle) cells; lowered levels of insulin cause liver cells to convert glycogen to glucose and excrete it into the blood.
Increased fatty acid synthesis – insulin facilitates the entry of blood lipids to adipose tissue which can then be converted to triglycerides; lack of insulin causes the reverse.
Decreased proteinolysis – forces reduction of protein degradation; lack of insulin increases protein degradation.
Decreased lipolysis – forces reduction in conversion of fat cell lipid stores into plasma fatty acids; lack of insulin causes the reverse.
Decreased gluconeogenesis – decreases production of glucose from various substrates in liver; lack of insulin causes glucose production from assorted substrates in the liver and elsewhere.
Increased amino acid uptake – facilitates the absorption of circulating amino acids; lack of insulin inhibits absorption.
Increased potassium uptake – forces cells to absorb serum potassium; lack of insulin inhibits absorption.
Arterial muscle tone – forces arterial wall muscle to relax, increasing blood flow, especially in micro arteries; lack of insulin reduces flow by allowing these muscles to contract.
The above excerpts are from various papers I’ve written and works by other authors I’ve reference in the past. I bring forth these items in response to the ever loudening clamor on the internet message boards in regard to what I can only term as ”glucaphobia”…….the irrational fear of sugar!
I’ve not written much on the subject of carbohydrate consumption and bodybuilding only because I felt it was a bit of a no brainer and to some degree an unworthy subject for an “EvilGenius”!
However for my own entertainment here is all you need to know to understand why you NEED sugar to achieve muscle building success!
Factual Statements
Athletes both professional and recreational are known to be taking exogenous insulin for its obvious and clinically documented ANABOLIC properties!
Sugars potentiate a potent insulin response
In child like simplicity!
INSULIN IS ANABOLIC
SUGAR STIMULATES INSULIN
THEREFORE
SUGAR IS ANABOLIC
In truth there is a bit more to the subject than just that, but not a whole lot more!
B.”EvilGenius”Chavez EvilGenius Sports Performance
www.EvilGSP.com
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